min bemondó Tapasztalt személy sp 600125 bab törékeny utal
JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine
The JNK inhibitor SP600125 enhances dihydroartemisinin-induced apoptosis by accelerating Bax translocation into mitochondria in human lung adenocarcinoma cells - ScienceDirect
SP600125 enhances C-2-induced cell death by the switch from autophagy to apoptosis in bladder cancer cells | Journal of Experimental & Clinical Cancer Research | Full Text
SP600125, an anthrapyrazolone inhibitor of Jun N-terminal kinase | PNAS
SP600125 | JNK Inhibitor | MedChemExpress
JNK inhibitor SP600125 protects against lipopolysaccharide‑induced acute lung injury via upregulation of claudin‑4
SP600125, an anthrapyrazolone inhibitor of Jun N-terminal kinase | PNAS
SP600125 enhances C-2-induced cell death by the switch from autophagy to apoptosis in bladder cancer cells | Journal of Experimental & Clinical Cancer Research | Full Text
JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine
JNK inhibitor SP600125 enhances TGF-β-induced apoptosis of RBE human cholangiocarcinoma cells in a Smad-dependent manner. | Semantic Scholar
Frontiers | SP600125, a JNK-Specific Inhibitor, Regulates in vitro Auricular Cartilage Regeneration by Promoting Cell Proliferation and Inhibiting Extracellular Matrix Metabolism
A c-Jun NH2-Terminal Kinase Inhibitor SP600125 (Anthra[1,9-cd]pyrazole-6 (2H)-one) Blocks Activation of Pancreatic Stellate Cells | Journal of Pharmacology and Experimental Therapeutics
JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis | Experimental & Molecular Medicine